Nov 2024, Volume 42 Issue 11
    

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    Editorial
  • Editorial
    Fazheng Ren,Xiaobin Wang,Bing Fang
  • News & Highlights
  • Mark Peplow
  • Chris Palmer
  • Mitch Leslie
  • Research
  • Yun Song Song, Zhang Hao, Guo Huiyuan, Wang Xiaobin

    The traditional approach with nutritional supplements is “one size fits all,” which has its limitations due to a lack of consideration of individual variations, including genetic background, health condition, lifestyle, and dietary intake. Depending on individual characteristics, the exact same nutritional supplementation could be beneficial, have no effect, or even be harmful. Precision nutrition has emerged as a new horizon, with the aim of carefully assessing individual nutritional and health needs and designing individually tailored diet and nutritional supplements. Engineering may play a significant role in each step of precision nutrition research and translation, including big data collection and management, nutritional and environmental assessment, data analyses, prediction, biomedical engineering, and nutritional engineering. The integration of precision nutrition and engineering could enhance our ability to best serve all people’s nutritional and health needs.

  • Tianshu Han,Wei Wei,Wenbo Jiang,Yiding Geng,Zijie Liu,Ruiming Yang,Chenrun Jin,Yating Lei,Xinyi Sun,Jiaxu Xu,Chen Juan,Changhao Sun
    The concept of precision nutrition was first proposed almost a decade ago. Current research in precision nutrition primarily focuses on comprehending individualized variations in response to dietary intake, with little attention being given to other crucial aspects of precision nutrition. Moreover, there is a dearth of comprehensive review studies that portray the landscape and framework of precision nutrition. This review commences by tracing the historical trajectory of nutritional science, with the aim of dissecting the challenges encountered in nutrition science within the new era of disease profiles. This review also deconstructs the field of precision nutrition into four key components: the proposal of the theory for individualized nutritional requirement phenotypes; the establishment of precise methods for measuring dietary intake and evaluating nutritional status; the creation of multidimensional nutritional intervention strategies that address the aspects of what, how, and when to eat; and the construction of a pathway for the translation and integration of scientific research into healthcare practices, utilizing artificial intelligence and information platforms. Incorporating these four components, this review further discusses prospective avenues that warrant exploration to achieve the objective of enhancing health through precision nutrition.
  • Shoujuan Yu,Ziyang Li,Xiaoxu Zhang,Qi Zhang,Liwei Zhang,Liang Zhao,Ping Liu,Jie Guo,Juan Chen,Chengying Zhang,Xinjuan Liu,Mengyang Yu,Dekui Jin,Xiaofeng Wang,Guang Li,Yan Cao,Fazheng Ren,Ran Wang
    Skin aging is an increasingly prominent topic in the context of healthy aging. During the aging process, the skin’s barrier function diminishes, its water content decreases, wrinkles begin to form, and changes occur in the gut microbiota composition. However, the relationship between gut microbiota and skin aging remains unclear. In this study, we explored skin rejuvenation in aged mice through fecal microbiota transplantation (FMT) using feces from young mice. The results demonstrated enhanced water retention, thickened stratum corneum, increased collagen content, and improved epithelial cell differentiation in aged mice following FMT. Notably, FMT particularly increased the abundance of Lactobacillus and Lactococcus in aged mice, which were nearly undetectable in untreated aged mice. Non-targeted and targeted metabolomics analyses indicated that FMT significantly elevated levels of tryptophan (Trp) and its microbiota metabolites (e.g., indole-3-lactic acid (ILA)) in the feces and serum of aged mice. Both Trp and ILA appeared to rejuvenate aged skin by activating the aryl hydrocarbon receptor (AhR) to promote epidermal cell differentiation. In conclusion, FMT from young mice rejuvenated aged skin via Trp-metabolizing bacteria (Lactobacillus and Lactococcus) and Trp-derived metabolites, suggesting that interventions targeting Trp metabolites may effectively improve skin aging.
  • Article
    Yingying Lin,Yao Lu,Yuqi Wang,Cong Lv,Juan Chen,Yongting Luo,Heng Quan,Weiru Yu,Lining Chen,Ziyu Huang,Yanling Hao,Qingyu Wang,Qingfeng Luo,Jingyu Yan,Yixuan Li,Wei Zhang,Min Du,Jian He,Fazheng Ren,Huiyuan Guo
    Intestinal stem cells (ISCs) initiate intestinal epithelial regeneration and tumorigenesis, and they experience rapid refilling upon various injuries for epithelial repair as well as tumor reoccurrence. It is crucial to reveal the mechanism underlying such plasticity for intestinal health. Recent studies have found that metabolic pathways control stem cell fate in homeostasis, but the role of metabolism in the regeneration of ISCs after damage has not been clarified. Here, we find that in a human colorectal cancer dataset, miR-29a and b (miR-29a/b) are metabolic regulators highly associated with intestinal tumorigenesis and worse prognostic value of radiotherapy. We also show that these two microRNAs are required for intestinal stemness maintenance in mice, and their expression is induced in regenerated ISCs after irradiation injury, resulting in skewed ISC fate from differentiation towards self-renewal. This upregulation of miR-29a/b expression in ISCs leads to suppression of fatty acid oxidation (FAO) and depression of oxidative phosphorylation, which in turn controls the balance between self-renewal and differentiation of ISCs. Deletion of miR-29a/b prevents these effects and thus impairs ISC-mediated epithelial recovery. Finally, we filter the potential targets of miR-29a/b and identify Hnf4g, a transcription factor, that drives this metabolic reprogramming through regulating FAO-related enzymes. Our work discovers an important metabolic mechanism of ISC-mediated regeneration and potentially pave the way for more targeted and effective therapeutic strategies for intestinal repair as well as tumor treatment.
  • Jianmin Wu,Ming Zhang,Hao Zhang,Mingxuan Sheng,Jiazeng Sun,Fang Wu,Haina Gao,Lishui Chen,Zhili Li,Qiyu Tian,Longjiao Zhu,Bing Fang
    Susceptibility to pathogens in the elderly is heightened with age, largely because of immunosenescence. As an immune regulatory organ, bone marrow creates immune cells that move to other organs and tissues through the blood. Despite the significance of this process of this organ, there is limited research on changes in immune cell generation in the bone marrow and their effects on immunosenescence. In this study, the compositions of immune cells in bone marrow from young (three months) and old (24+ months) mice were compared by means of mass cytometry, with further validation obtained through the reanalysis of single-cell RNA sequencing data and cell sorting via flow cytometry. The effects of differential immune cells on immunosenescence in old mice were evaluated using the Clostridioides difficile (C. difficile) infection model. Our results showed that aged mice presented with a reduction in bone trabeculae structure, which was accompanied by a notable increase in polymorphonuclear (PMN)-myeloid-derived suppressor cell (MDSC) abundance. Through bulk-seq and reverse transcription quantitative polymerase chain reaction (RT-qPCR) analysis, we identified differential genes associated with the immune response—specifically, the Th17 cell differentiation pathway. Furthermore, the increase in exported PMN-MDSCs to the large intestine resulted in increased gut permeability and inflammatory damage to the colon following C. difficile infection. After clearing the PMN-MDSCs in old mice using the anti-Gr-1 antibody, the symptoms induced by C. difficile were significantly relieved, as evidenced by an inhibited IL-17 pathway in the colon and reduced gut permeability. In conclusion, aging increases the number of PMN-MDSCs in both the generated bone marrow and the outputted intestine, which contributes to susceptibility to C. difficile infection. This study provides a novel target for anti-aging therapy for immunosenescence, which is beneficial for improving immune function in elders.
  • Rui Quan,Chenhong Shi,Yanan Sun,Chengying Zhang,Ran Bi,Yiran Zhang,Xin Bi,Bin Liu,Ziheng Dong,Dekui Jin,Yixuan Li
    Pulmonary fibrosis (PF) is a lethal lung disease that predominantly affects older adults; however, whether and how aging triggers fibrosis remains unclear. To pinpoint the predominant initiating factors of PF, we first analyzed single-cell RNA sequencing (scRNA-seq) data from the lung tissues of 45 normal donors and 51 PF patients and found that aging might serve as the primary catalyst for PF development. To further investigate the influence of aging on PF formation, we conducted a comprehensive and thorough study employing a natural aging mouse model. We found that dynamic alterations in the quantity and types of collagen fibers during aging-induced PF progression, especially in Col I, emerged as the predominant driver of PF. We then investigated the regulation of Col I synthesis during aging using primary alveolar type 2 (AT2) cells and A549 cells line through conditioned media and Transwell coculture, and found that secretions—particularly PAI-1—from aged AT2 cells promoted fibrosis and enhanced Col1al production via the TGF-β/Smad2/3 pathway. Furthermore, scRNA-seq and a histological analysis of human lung tissue demonstrated a significant upregulation of SERPINE1 (the gene encoding PAI-1) and PAI-1 expression in both aging lung tissue and AT2 cells, which was consistent with our findings from animal experiments, providing additional evidence for the pivotal role of PAI-1 during aging and the development of PF. Our research demonstrates that PAI-1, a crucial factor secreted by aging AT2 cells, exerts a pivotal role in promoting the synthesis of Col1a1 in fibroblasts, subsequently leading to Col I deposition, and in driving the progression of PF by mediating the TGF-β/Smad2/3 pathway. Our findings offer critical evidence for the involvement of epithelial dysfunction in age-related PF and provides potential novel therapeutic targets for clinical intervention.
  • Rui Song,Guang Li,Liang Zhao,Lili Qiu,Xiyu Qin,Xiaoxu Zhang,Xiaoxue Liu,Jun Zhou,Mengxiao Hu,Liwei Zhang,Jiaqi Su,Xinjuan Liu,Xiaoyu Wang
    As the global demographic shifts toward an aging population, understanding the efficiency of protein utilization in older adults becomes crucial. Our study explores the intricate relationship between protein intake and aging, with a focus on precision nutrition for older people. Through a meta-analysis, we confirm a decline in protein-utilization capacity in older individuals and examine the different contributions of plant and animal protein. In experiments involving mice of different ages, older mice exhibited decreases in the biological utilization of four proteins (casein, beef protein, soy protein, and gluten), particularly casein. In subsequent research, casein was studied as a key protein. A decline in gastric digestion function was observed through peptidomics and the examination of pepsin levels using casein. Nevertheless, this decline did not significantly affect the overall protein digestion during the aging process. The combined application of targeted amino acid metabolomics identified abnormal absorption of amino acids as the underlying cause of decreased protein utilization during aging, particularly emphasizing a reduction in branched-chain amino acids (BCAAs) in older mice. Delving deeper into the proteomics of the intestinal protein digestion and absorption pathway, a reduction of over 60% in large neutral amino acid transporter 2 (LAT2) protein expression was observed in both older humans and aged mice. The reduction in LAT2 protein was found to be a key factor influencing the diminished BCAA availability. Overall, our study establishes the significance of amino acid absorption through LAT2 in protein utilization during aging and offers a new theoretical foundation for improving protein utilization in the older adults.
  • Peng An,Sitong Wan,Langrun Wang,Tiancheng Xu,Teng Xu,Yonghui Wang,Jin Liu,Keji Li,Xifan Wang,Jingjing He,Simin Liu
    The inconsistent findings concerning the effects of vitamin D supplementation on cardiometabolic risk factors and the large heterogeneity in the published literature call for further research to identify sources of heterogeneity and potential effect modifiers. We performed a meta-analysis of randomized controlled trials (RCTs) published until March 2024 that reported estimates for the effects of vitamin D supplementation on cardiometabolic factors and relevant baseline covariates of RCT participants. A total of 17 656 participants from 99 RCTs were analyzed, and weighted mean differences (95% confidence intervals (CI)) for the intervention status were derived using random-effects modeling. Overall, compared with the placebo, vitamin D supplementation (median dose: 3320 IU·day−1; range 40–120 000 IU·day−1) had favorable effects on systolic blood pressure (SBP; −2.04 (−3.50, −0.59) mmHg; 1 mmHg = 0.133 kPa), diastolic blood pressure (DBP; −3.00 (−3.61, −2.39) mmHg), total cholesterol (TC; −0.12 (−0.21, −0.03) mmol·L−1), fasting blood glucose (FBG; −0.13 (−0.20, −0.05) mmol·L−1), hemoglobin A1C (A1C; −0.09 (−0.13, −0.05)%), and fasting blood insulin (FBI: −7.61 (−11.93, −3.30) pmol·L−1). The benefits of vitamin D were most evident in trials performed in non-Westerners, participants with baseline 25-hydroxyvitamin D (25[OH]D) lower than 15.0 ng·mL−1, non-obese (body mass index (BMI) < 30 kg·m−2), and older (age ≥ 50 years). The findings of this study underscore the need for personalized vitamin D intervention strategies that comprehensively account for individual patient characteristics (such as ethnocultural background, age, BMI, and circulating 25[OH]D level), intervention dosage, and intervention duration to optimize cardiometabolic health outcomes.
  • Xifan Wang,Pengjie Wang,Yixuan Li,Huiyuan Guo,Ran Wang,Siyuan Liu,Ju Qiu,Xiaoyu Wang,Yanling Hao,Yunyi Zhao,Haiping Liao,Zhongju Zou,Josephine Thinwa,Rong Liu
    Inflammatory bowel disease (IBD) refers to a pair of prevalent conditions (Crohn’s disease and ulcerative colitis) distinguished by persistent inflammation of the large intestine. Procyanidin C1 (PCC1) is a naturally occurring substance derived from grape seeds that has demonstrated notable anti-inflammatory properties. This study examines the potential utility of PCC1 as a treatment for IBD and subsequently examines the host-cell- and microbiome-related mechanisms underlying the detected therapeutic benefits. Working with a classic dextran sodium sulfate (DSS)-induced mouse IBD model, we show that PCC1 protects the mucosal barrier and thereby confers strong protective effects against IBD. PCC1 pretreatment resulted in anti-inflammatory effects and protection against multiple pathological phenotypes in the IBD model mice, including reduced weight loss, lower Disease Activity Index (DAI) totals, and enhanced colon size, as well as obviously beneficial effects on the mucosal barrier (e.g., barrier thickness and activity of mucus-degrading enzymes). We also analyzed the autophagy marker LC3 and found that the level of LC3 was significantly elevated in the intestinal epithelial cell samples of the PCC1-pretreatment group as compared with the non-model mice samples. PCC1 altered the fecal microbiome composition, which included elevating the abundance of Akkermansia muciniphila and Christensenella minuta. Fecal microbiome transplant (FMT) experiments showed that delivering a microbiome from PCC1-treated animals into PCC1-naïve animals conferred protection. Metabolic profiling revealed that both the PCC1-pretreatment and PCC1 FMT groups had elevated levels of the microbiota-derived metabolite valeric acid, and supplementation with this short-chain fatty acid (SCFA) also conferred strong protection against IBD. Finally, inhibitor experiments confirmed that the beneficial effects of valeric acid on the mucus layer are mediated by FOXO1 signaling in the goblet cells of the intestinal epithelium. Beyond showing that PCC1 confers anti-inflammatory effects and protection against IBD by altering the microbiome, our study demonstrates proof of principle for multiple straightforward interventions (PCC1, FMT, and valeric acid supplementation) for ameliorating mucosal barrier damage to treat IBD.
  • Xiang Wang,Di Zhang,Yogendra Pratap Singh,Miji Yeo,Guotao Deng,Jiaqi Lai,Fei Chen,Ibrahim T. Ozbolat,Yin Yu
    Organ damage or failure arising from injury, disease, and aging poses challenges due to the body’s limited regenerative capabilities. Organ transplantation presents the issues of donor shortages and immune rejection risks, necessitating innovative solutions. The 3D bioprinting of organs on demand offers promise in tissue engineering and regenerative medicine. In this review, we explore the state-of-the-art bioprinting technologies, with a focus on bioink and cell type selections. We follow with discussions on advances in the bioprinting of solid organs, such as the heart, liver, kidney, and pancreas, highlighting the importance of vascularization and cell integration. Finally, we provide insights into key challenges and future directions in the context of the clinical translation of bioprinted organs and their large-scale production.
  • Xu Chen,Zhiyong Du,Dongqing Guo,Jincheng Guo,Qianbin Sun,Tiantian Liu,Kun Hua,Chun Li,Yong Wang,Wei Wang
    This investigation elucidates the spatiotemporal dynamics of NOD-like receptor family pyrin domain containing 3 (NLRP3) inflammasome activation following myocardial infarction (MI), a process that has not been fully characterized. We revealed early activation of the NLRP3 inflammasome in mice with MI and characterized its dynamic temporal expression. Notably, the knockout and inhibition of Nlrp3 expression were found to significantly mitigate infarct size and enhance cardiac function. Furthermore, our analysis of the spatial characteristics of inflammasome activation revealed predominant activation in macrophages and subsequent activation in fibroblasts on the third day post-MI. To elucidate the nexus between macrophage-associated NLRP3 inflammasome activation and myocardial fibrosis, we employed targeted metabolomics analyses of inflammatory oxylipins, small interfering RNA (siRNA) interference experiments, and various molecular assays. These findings revealed that macrophage-associated inflammasome activation facilitates the conversion of fibroblasts into myofibroblasts via the 15-hydroxy-5,8,11,13-eicosatetraenoic acid (15-HETE)-mediated small mother against decapentaplegic (Smad) pathway. Additionally, both mass spectrometry imaging (MSI) and targeted metabolomics analyses confirmed the significant increase in 15-HETE levels in mice with MI and in patients with MI and acute coronary syndrome (ACS). Our comprehensive dataset suggests that NLRP3 inflammasome activation in MI is characterized by distinct temporal and spatial patterns. These insights mark a significant advancement toward precise MI prevention and treatment strategies, particularly early myocardial fibrosis intervention.
  • Ya Gao,Peng Yin,Haidong Kan,Renjie Chen,Maigeng Zhou
    Stock volatility constitutes an adverse psychological stressor, but few large-scale studies have focused on its impact on major adverse cardiovascular events (MACEs) and suicide. Here, we conducted an individual-level time-stratified case-crossover study to explore the association of daily stock volatility (daily returns and intra-daily oscillations for three kinds of stock indices) with MACEs and suicide among more than 12 million individual decedents from all counties in the mainland of China between 2013 and 2019. For daily stock returns, both stock increases and decreases were associated with increased mortality risks of all MACEs and suicide. There were consistent and positive associations between intra-daily stock oscillations and mortality due to MACEs and suicide. The excess mortality risks occurred at the current day (lag 0 d), persisted for two days, and were greatest for suicide and hemorrhagic stroke. Taking the present-day Shanghai and Shenzhen 300 Index as an example, a 1% decrease in daily returns was associated with 0.74%–1.04% and 1.77% increases in mortality risks of MACEs and suicide, respectively; the corresponding risk increments were 0.57%–0.85% and 0.92% for a 1% increase in daily returns and 0.67%–0.77% and 1.09% for a 1% increase in intra-daily stock oscillations. The excess risks were more pronounced among individuals aged 65–74 years, males, and those with lower education levels. Our findings revealed considerable health risks linked to sociopsychological stressors, which are helpful for the government and general public to mitigate the immediate cardiovascular and mental health risks associated with stock market volatility.
  • Article
    Author links open overlay panel Xiaoxuan Zhang, Hanxu Chen, Taiyu Song, Jinglin Wang, Yuanjin Zhao

    Investigation of patient-derived primary tissues is of great importance in the biomedical field, but recent tissue slicing and cultivation techniques still have difficulties in satisfying clinical requirements. Here, we propose a controllable histotomy strategy that utilizes hierarchical magnetic microneedle array robots to tailor primary tissues and establish the desired high-throughput tissue-on-a-chip. This histotomy is performed using a three-dimensional printed, mortise-tenon-structured slicing device coupled with a magnetic-particle-loaded and pagoda-shaped microneedle array scaffold. Due to the multilayered structure of these microneedles, tissue specimens can be fixed onto the microneedle scaffold via mechanical interlocking, thereby effectively avoiding tissue slipping during the slicing process. Owing to the encapsulation of magnetic microneedle fragments, these tissue pieces can act as magnetically responsive biohybrid microrobots and can be easily manipulated by magnetic fields, facilitating their separation, transportation, and dynamic culture. Using this strategy, we demonstrate that primary pancreatic cancer tissues can be tailored into tiny pieces and cultured in multilayered microfluidic chips for efficient high-throughput drug screening, indicating the promising future of this technique’s application in clinical settings.

  • Weiliang Wen,Shenghao Gu,Ying Zhang,Wanneng Yang,Xinyu Guo
    Crop phenomics has rapidly progressed in recent years due to the growing need for crop functional genomics, digital breeding, and smart cultivation. Despite this advancement, the lack of standards for the creation and usage of crop phenomics technology and equipment has become a bottleneck, limiting the industry’s high-quality development. This paper begins with an overview of the crop phenotyping industry and presents an industrial mapping of technology and equipment for big data in crop phenomics. It analyzes the necessity and current state of constructing a standard framework for crop phenotyping. Furthermore, this paper proposes the intended organizational structure and goals of the standard framework. It details the essentials of the standard framework in the research and development of hardware and equipment, data acquisition, and the storage and management of crop phenotyping data. Finally, it discusses promoting the construction and evaluation of the standard framework, aiming to provide ideas for developing a high-quality standard framework for crop phenotyping.
  • Article
    Runxi Wang,Lu Qiao,Mufei Liu,Yanpeng Ran,Jun Wang,Wupeng Yan,Yan Feng,Li Cui
    Sugar aminotransferases (SATs) catalyze the installation of chiral amines onto specific keto sugars, producing bioactive amino sugars. Their activity has been utilized in artificial reactions, such as using the SAT WecE to transform valienone into the valuable α-glucosidase inhibitor valienamine. However, the low thermostability and limited activity on non-natural substrates have hindered their applications. Simultaneously improving stability and enzyme activity is particularly challenging owing to the acknowledged inherent trade-off between stability and activity. A customized combinatorial active-site saturation test-iterative saturation mutagenesis (CAST-ISM) strategy was used to simultaneously enhance the stability and activity of WecE toward valienone. Fourteen hotspots related to improving the stability-activity trade-off were identified based on evolutionary conservation and the average mutation folding energy assessment of 57 residues in the active site of WecE. Positive mutagenesis and combinatorial mutations of these specific residues were accomplished via site-directed saturation mutagenesis (SSM) and iterative evolution cycles. Compared with those of the wild-type (WT) WecE, the quadruple mutant M4 (Y321F/K209F/V318R/ F319V) displayed a 641.49-fold increase in half-life at 40 °C and a 31.37-fold increase in activity toward the non-natural substrate valienone. The triple mutant M3 (Y321F/K209F/V318R) demonstrated an 83.04-fold increase in half-life at 40 °C and a 37.77-fold increase in activity toward valienone. The underlying mechanism was dependent on the strengthened interface interactions and shortened transamination reaction catalytic distance, compared with those of the WT, which improved the stability and activity of the obtained mutants. Thus, we accomplished a general target-oriented strategy for obtaining stable and highly active SATs for artificial amino-sugar biosynthesis applications.
  • Article
    Hong-Yun Zhang, Wei-Dan Jiang, Pei Wu, Yang Liu, Hong-Mei Ren, Xiao-Wan Jin, Xiao-Qiu Zhou, Lin Feng

    Zearalenone (ZEA), a mycotoxin, poses a significant global hazard to human and animal health. Natural products (NPs) have shown promise for mitigating the adverse effects of ZEA owing to their diverse functional activities. However, the current challenge lies in the absence of an efficient strategy for systematic screening and identification of NPs that can effectively protect against ZEA-induced toxicity. This study describes a phenotype-based screening strategy for screening NP libraries and discovering more effective compounds to mitigate or counteract the adverse consequences of ZEA exposure in animals. Using this strategy, we initially identified 96 NPs and evaluated the potency and efficacy of two effective candidate compounds, fraxetin, and hydroxytyrosol, based on embryonic phenotype and locomotor activity using a scoring system and the TCMacro method. Furthermore, we performed transcriptome and protein−protein interaction (PPI) network analyses to extract two mRNA signatures to query the Connectivity Map (CMap) database and predict NPs. The predicted NPs showed the potential to reverse the gene expression profiles associated with ZEA toxicity. Consequently, we further screened these compounds using our model, which indicated that hispidin, daphnetin, and riboflavin exhibit promising in vivo efficacy in zebrafish. Notably, throughout the process, fraxetin consistently stood out as the most promising NP. Biological pathway analysis and functional verification revealed that fraxetin completely reversed the toxic effects of ZEA at very low doses. This was achieved by repairing damaged cell apoptosis, modifying the cell cycle pathway, and preventing senescence induction, indicating good application potential. Overall, we demonstrated that this integration strategy can be successfully applied to effectively discover potential antidotes.

  • Zong Lu,Haoyu Wu,Yanying Wei,Haihui Wang
    Transport channels with ultrahigh K+ selectivity over other ions play a crucial role for living beings, but constructing ionic channels with promising K+ selectivity and permeability remains a challenge. Here, an asymmetric bilayer membrane based on MXene (Ti3C2Tx) lamellar channels consisting of a recognition layer (RL) on top of an enhancement layer (EL) exhibits an amazing Matthew effect: amplification of the preferred transport of K+, resulting in an excellent K+-separation performance. The K+ ion is selected by the 1-aza-18-crown-6 ether-modified RL, owing to preferential affinity energy, and then rapidly transported as a hydrated ion through the EL, based on the confinement effect. Other undesired ions such as Na+ are hindered from entering the RL by the preferred K+ occupation of the crown ether. The MXene (Ti3C2Tx)-based Matthew membrane presents high K+-permeation rates of 0.1–0.2 mol∙m–2∙h–1, with a significant K+/Na+ selectivity of 5–9. The molecular separation mechanism of the Matthew membrane is investigated deeply to explore the nature of the Matthew amplification effect on K+ sieving, where the precise matching of the RL and EL within the membrane governs the fast K+ permeation with good selectivity. The asymmetric structure of our Matthew membrane is the key to understanding the biological function of ion channels for precise and fast ion transport, which will guide us in the creation of artificial ion channels or membranes.
  • Shu Tian,Jinli Zhang,Shuan Liu,Jingyu Li,Jibin Pu,Yugang Hao,Guobing Ying,Qunji Xue,Guangming Lu
    Marine corrosion and biofouling are challenges that affect marine industrial equipment, and protecting equipment with functional coatings is a simple and effective approach. However, it is extremely difficult to combine anti-corrosion and anti-fouling properties in a single coating. In this work, we combine reduced graphene oxide (rGO)/silver nanoparticles (AgNPs) with a hydrophilic polymer in a bio-based silicone–epoxy resin to create a coating with both anti-fouling and anti-corrosion properties. The excellent anti-fouling performance of the coating results from a ternary synergistic mechanism involving fouling release, contact inhibition, and a hydration effect, while the outstanding anti-corrosion performance is provided by a ternary synergistic anti-corrosion mechanism that includes a dense interpenetrating network (IPN) structure, a barrier effect, and passivation. The results show that the obtained coating possesses superior anti-fouling activity against protein, bacteria, algae, and other marine organisms, as well as excellent anti-corrosion and certain self-healing properties due to its dynamic cross-linked network of rGO/AgNPs and the hydrophilic polymer. This work provides an anti-corrosion and anti-fouling integrated coating for marine industrial equipment.
  • Huaiyu Wu,Jinwook Kim,Bohua Zhang,Gabe Owens,Greyson Stocker,Mengyue Chen,Benjamin C. Kreager,Ashley Cornett,Kathlyne Bautista,Tarana Kaovasia,Paul A. Dayton,Zhen Xu,Xiaoning Jiang
    Thromboembolism in blood vessels poses a serious risk of stroke, heart attack, and even sudden death if not properly managed. Sonothrombolysis combined with ultrasound contrast agents has emerged as a promising approach for the effective treatment of thromboembolism. Recent reports have highlighted the potential of intravascular sonothrombolysis as a safe and effective treatment modality for deep vein thrombosis (DVT). However, its efficiency has not been validated through in vivo testing of retracted clots. This study aimed to develop a miniaturized multidirectional transducer featuring two 4-layer lead zirconate titanate (PZT-5A) stacks with an aperture size of 1.4 mm × 1.4 mm, enabling both forward- and side-looking treatment. Integrated into a custom two-lumen 10-French (Fr) catheter, the capability of this device for intravascular sonothrombolysis was validated both in vitro and in vivo. With low-dose tissue plasminogen activators and nanodroplets, the rotational multidirectional transducer reduced the retracted clot mass (800 mg) by an average of 52% within 30 min during in vitro testing. The lysis rate was significantly higher by 37% than that in a forward-viewing transducer without rotation. This improvement was particularly noteworthy in the treatment of retracted clots. Notably, a long-retracted clot (> 10 cm) was successfully treated within 40 min in vivo by creating a flow channel with a diameter > 4 mm in a porcine DVT model. In conclusion, these findings strongly suggest the potential of this technique for clinical applications in sonothrombolysis, offering a feasible solution for effectively treating thromboembolism, particularly in challenging cases involving retracted clots
  • Daxin Tian,Jianshan Zhou,Xu Han,Ping Lang
    Obstacle detection and platoon control for mixed traffic flows, comprising human-driven vehicles (HDVs) and connected and autonomous vehicles (CAVs), face challenges from uncertain disturbances, such as sensor faults, inaccurate driver operations, and mismatched model errors. Furthermore, misleading sensing information or malicious attacks in vehicular wireless networks can jeopardize CAVs’ perception and platoon safety. In this paper, we develop a two-dimensional robust control method for a mixed platoon, including a single leading CAV and multiple following HDVs that incorporate robust information sensing and platoon control. To effectively detect and locate unknown obstacles ahead of the leading CAV, we propose a cooperative vehicle–infrastructure sensing scheme and integrate it with an adaptive model predictive control scheme for the leading CAV. This sensing scheme fuses information from multiple nodes while suppressing malicious data from attackers to enhance robustness and attack resilience in a distributed and adaptive manner. Additionally, we propose a distributed car-following control scheme with robustness to guarantee the following HDVs, considering uncertain disturbances. We also provide theoretical proof of the string stability under this control framework. Finally, extensive simulations are conducted to validate our approach. The simulation results demonstrate that our method can effectively filter out misleading sensing information from malicious attackers, significantly reduce the mean-square deviation in obstacle sensing, and approach the theoretical error lower bound. Moreover, the proposed control method successfully achieves obstacle avoidance for the mixed platoon while ensuring stability and robustness in the face of external attacks and uncertain disturbances.
  • Article
    Junping Gu,Xiaoyu Qian,Yiwei Liu,Qinggong Wang,Yiyang Zhang,Xuan Ruan,Xiangjin Deng,Yaowen Lu,Jian Song,Hui Zhang,Yunning Dong,Mengmeng Wei,Wei Yao,Shuiqing Li,Weihua Wang,Zhigang Zou,Mengfei Yang
    Facing the challenges of in-situ utilization of lunar regolith resources, applying an external electric field to manipulate lunar particles has become a promising method for space particle control, which mainly depends on the particle charging properties in the applied electric field. Using the surficial lunar regolith samples brought back from the Moon by the Chang’e-5 mission (CE5 LS), this work studies their charging properties, particle dynamics, and their collision damage to aerospace materials under the action of an external electric field in high-vacuum conditions. The results show that the charging process and electrostatic projection of lunar regolith particles under high-vacuum conditions were different from those under atmosphere conditions. The particle diameter range of CE5 LS used in the experiment is 27.7–139.0 μm. For electric field strength of 3–12 kV·cm−1, the charge obtained by CE5 LS is 4.8 × 10−15–4.7 × 10−13 C and the charge-to-mass ratio is 1.2 × 10−5–6.8 × 10−4 C·kg−1. The CE5 LS is easier to be negatively charged in an external electric field. Furthermore, significant damages were observed on the target impact surfaces, indicating severe influences of lunar regolith particles on aerospace materials. Our work provides a deeper understanding of fundamental principles for lunar regolith shielding and utilization, and will inspire broad efforts to develop the lunar in-situ engineering solutions.
  • Review
    Caineng Zou,Zhengfu Zhao,Songqi Pan,Jia Yin,Guanwen Lu,Fangliang Fu,Ming Yuan,Hanlin Liu,Guosheng Zhang,Cui Luo,Wei Wang,Zhenhua Jing
    The lower Cambrian Qiongzhusi (Є1q) shale in the Sichuan Basin, formerly considered a source rock, recently achieved high gas production (7.388 × 105 m3·d−1) from well Z201 in the Deyang–Anyue rift trough (DART), marking an exploration breakthrough of the world’s oldest industrial shale gas reservoir. However, the shale gas enrichment mechanism within the DART is not fully understood. This study reviews the formation of the Qiongzhusi shale gas reservoirs within the DART by comparing them with cotemporaneous deposits outside the DART, and several findings are presented. The gas production interval was correlated with the main phase of the Cambrian explosion (lower Cambrian stage 3). In the early Cambrian ecosystem, dominant animals likely accelerated the settling rates of organic matter (OM) in the upper 1st member of Є1q (Є1q12) by feeding on small planktonic organisms and producing larger organic fragments and fecal pellets. High primary productivity and euxinic conditions contributed to OM enrichment in the lower 1st member of Є1q (Є1q11). Additionally, shale reservoirs inside the DART demonstrated better properties than those outside in terms of thickness, brittle minerals, gas content, and porosity. In particular, the abundant OM pores inside the DART facilitated shale gas enrichment, whereas the higher thermal maturity of the shales outside the DART possibly led to the graphitization and collapse of some OM pores. Meanwhile, the overpressure of high-production wells inside the DART generally reflects better shale gas preservation, benefiting from the shale’s self-sealing nature, “upper capping and lower plugging” configuration, and limited faults and microfractures. Considering these insights, we introduced a “ternary enrichment” model for the Qiongzhusi shale gas. Although the current high gas production of Z201 was found at the reservoir 3, two additional reservoirs were identified with significant potential, thus suggesting a “multilayer stereoscopic development” strategy in future shale gas exploration within the DART.
  • Qi Zhou,Shen Qu,Miaomiao Liu,Jianxun Yang,Jia Zhou,Yunlei She,Zhouyi Liu,Jun Bi
    Top-down environmental policies aim to mitigate environmental risks but inevitably lead to economic losses due to the market entry or exit of enterprises. This study developed a universal dynamic agent-based supply chain model to achieve tradeoffs between environmental risk reduction and economic sustainability. The model was used to conduct high-resolution daily simulations of the dynamic shifts in enterprise operations and their cascading effects on supply chain networks. It includes production, consumption, and transportation agents, attributing economic features to supply chain components and capturing their interactions. It also accounts for adaptive responses to daily external shocks and replicates realistic firm behaviors. By coupling high spatial-temporal resolution firm-level data from 18 916 chemical enterprises, this study investigates the economic and environmental impacts of an environmental policy resulting in the closure of 1 800 chemical enterprises over three years. The results revealed a significant economic loss of 25.8 billion USD, ranging from 23.8 USD to 31.8 billion USD. Notably, over 80% of this loss was attributed to supply chain propagation. Counterfactual analyses indicated that implementing a staggered shutdown strategy prevented 18.8% of supply chain losses, highlighting the importance of a gradual policy implementation to prevent abrupt supply chain disruptions. Furthermore, the study highlights the effectiveness of a multi-objective policy design in reducing economic losses (about 29%) and environmental risks (about 40%), substantially enhancing the efficiency of the environmental policy. The high-resolution simulations provide valuable insights for policy designers to formulate strategies with staggered implementation and multiple objectives to mitigate supply chain losses and environmental risks and ensure a sustainable future.
  • Article
    Tiejun Liu, Ming Zhang, Dujian Zou, Jiaping Liu, Jinping Ou

    Concerns about the durability of transportation infrastructure due to freeze-thaw (F-T) cycles are particularly significant in the Chinese plateau region, where concrete aging and performance deterioration pose substantial challenges. The current national standards for the frost resistance design of concrete structures are based predominantly on the coldest monthly average temperature and do not adequately address the comprehensive effects of the spatiotemporal variance, amplitude, and frequency of F-T cycles. To address this issue, this study introduced a spatiotemporal distribution model to analyze the long-term impact of F-T action on concrete structures by employing statistical analysis and spatial interpolation techniques. Cluster analysis was applied to create a nationwide zonation of F-T action level from data on the freezing temperature, temperature difference, and the number of F-T cycles. Furthermore, this study explored the similarity between natural environmental conditions and laboratory-accelerated tests using hydraulic pressure and cumulative damage theories. A visualization platform that incorporates tools for meteorological data queries, environmental characteristic analyses, and F-T action similarity calculations was designed. This research lays theoretical groundwork and provides technical guidance for assessing service life and enhancing the quantitative durability design of concrete structures in the Chinese plateau region.

  • Shihao Xiao,Limin Zhang,Te Xiao,Ruochen Jiang,Dalei Peng,Wenjun Lu,Xin He
    The 2280 km long Jinsha River has been blocked at least four times in the past 30 years. A landslide damming hazard chain can endanger communities and infrastructures hundreds of kilometers downstream from the damming site in alpine gorges. Past damming events have resulted in severe consequences, demanding a thorough assessment of damming threats along the entire Jinsha River. This study digitizes the Jinsha River and visualizes its topographic, tectonic, hydrologic, and climate characteristics in detail. A two-stage full-probability method is proposed for assessing the damming threats along this river, making it possible to identify potential damming hotspots and high-priority zones for hazard mitigation. It is found that the upper reach of the Jinsha River poses the greatest damming threat, and the threat level gradually decreases downstream. Approximately 33.4%, 36.7%, 20.5%, and 9.4% of the entire length of the Jinsha River are classified as low, moderate, high, and very high threat levels, respectively. Compared with existing hydropower projects, future projects in the upper reach are more likely to be exposed to landslide damming. We highlight the value of basin-scale spatial threat analysis and envisage that our findings will promote more targeted local-scale risk assessments for potential damming hotspots. These outcomes provide the basis for managing the risks of river damming and hydropower infrastructure along the Jinsha River.